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torsdag 4 juni 2015

1996 Free Paper Abstracts. Clinics. A28- A46

A28
AN INSTRUCTIONAL VIDEO ON THE MANAGEMENT OF CELIAC DISEASE
Molloy M, Davidson AGF, B.C:  Chapter of the Canadian Celiac Association. Vancouver BC , Canada
Feedback regarding the video has been very positive and supports the premise that video is an effective medium for isntruction or trnafer of information.  
 http://www.powershow.com/view/3b1b30-NTk4M/Celiac_Sprue_Review_of_a_Multisystem_Disease_Thomas_Repas_powerpoint_ppt_presentation

A29
CEREAL INTAKE OF ESTONIAN INFANTS
Mitt K, Uibo O. Tartu, Estonia 

A30
GLUTEN FREE DIET (GFD);: EFFECTS ON THE GROWTH PROGNOSIS OF COELIAC CHILDREN DIAGNOSED LATELY AND FOLLOWED AT LONG TERM
 Boudraa G, Benbouabdellah M, Moussaoussaid S, Haddou A, Touhami M.  Oran. Algeria
Th CD screening should be made early and the CD follow-up should be prolonged after the adolescence because the statural gain is always possible. 

A31
PECULIARITIES OF COELIAC DISEASE IN CHILDREN OF THE URAL REGION
Bolotova MF, Koruykina LP, Surinov VA. Perm, Russia
Background: Noninfectious pathology of gastrointestinal tract in children is an actual problem. Coeliac disease diagnosis is difficult.  
The aim of our study was to investigate clinico-laboratory peculiarities of this disease  in children of Perm and region. 
Methods. Clinico-laboratory examination of 17 patients included general and biochemical analysis of blood with determination of protein fractions, lipogramme, electrolytes, enzymes: coprogramme, determination of trypsin and disacharide content in feces, estimation of intestinal flora, sweat test, T-, B-lymphocytes and immunoglobulins, USI of liver and spleen, bone roentgenography.  
 Results:  During the last 3 years coeliac disease was diagnostised in 17 children. The first symptoms appear, when children take gliadincontaining food: loose, butter stool (100%), body mass decrease. Arrest of physical development, xeroderma, muscle hypotension, anorexia, vomiting, abdominal pain were revealed in 70% of children, pseudoascites in 2, osteoporosis in 4 children. Half of children demonstrated hypochromic anemia of the I stage and eosinophilia.. Dysproteinemia was determined in 30%, cholesterol, beta-lipoprotein,  calcium decrease  in 50% . Fatty acids, neutral fat, muscle fibers, iodophilic bacteria, protein detritus were revealed in coprogramme. Disturbance of intestinal microflora correlation was estimated.  Decrease of theophyllinesensitive T-lymphicytes and IgA level was obtained in immunogramme in 1/3 of children. USI demonstrated pancreas changes in 50%. Prescription of agliadin diet and enzymes led to appetite improvement, stool normlization, body mass growth , anemia disappearance.



A32
UNCHANGED CLINICAL PATTERN OF CHILDHOOD COELIAC DISEASE IN SICILY
Cataldo F, Bottaro G, Traverso G, Violante M, Palermo, Catania, Italy

A33
A TYPICAL FORMS OF COELIAC DISEASE (CD),
Olazabal JI, Alvarez R, Ariza F, Ramos A., Loza C,  Asturias, Spain.

A34
PREVIOUS MISDIAGNOSIS AND DIAGNOSTIC DELAY IN COELIAC DISEASE
Brusco G, DiStefano M, Andreani ML, Bisgi F, Jorizzo RA, Arfilli L, Taglieri G, GAsbarrini G, Corazza GR, Italy 

A35
COELIAC DISEASE, A COMPARATIVE STUDY OF TWO PERIODS; 1975 TO 1984 AND 1985 TO  1994. 
Junqueira JC, Calcado AC, Percope S, Tao M.  Rio de Janeiro, Brazil

A36
COELIAC DISEASE IN THE ELDERLY: A MULTICENTRIC STUDY
DeVitis I, S´Addesa , Pasqualetti MR, Brusco G, Corazza GR, Gasbarrini G.  L-Aquila, Italy

A37
PATIENTS WITH SUBTOTAL VILLOUS ATROPHY: DIFFICULTIES OF MAKING ETHIOLOGICAL DIAGNOSIS AND FOLLOW UP
Tao MT, Junqueira JC, Carvalho CRF, MAfra CNC, Gracia J,,Calcado AC, Guerra SNRP. Rio De Janiero, Brazil 

A38
NUTRITIONAL STATUS OF NEWLY DIAGNOSED COELIAC PATIENTS- ASSOCIATION WITH THE GRADE OF VILLOUS ATROPHY
(Partial atrophy, Subtotal atrophy, Total villous atrophy) 
Kemppainen T, Kosma V-M, Janatuinen E, Julkunen R, uusitupa M.

A39
SUPERIOR MESENTERIC ARTERY BLOOD FLOW IN CELIAC CHILDREN 
Ertem, D, Tüney D, Pehlivanoglu E, Istanbul, Turkey

A40
ULTRASONOGRAPHY OF THE ABDOMEN IN CHILDREN WITH COELIAC DISEASE
Kuleta-Bosak E, Kalita B, Sikora A, Slimok M,, Urban K, Bytom, Poland. 
Ultrasonographic examination was carried out by means of Sonoline SL1, S1250 transducer sectoral 5 MHz and linear 5 and 7,5 MHz.   All the organs of the abdomen were examined and special attention was paid to the appearance of the loops of the intestine i.e. width, contents, speed of peristalsis. In all cases, serum was tested for IgA EmA/IgG EmA and in particular cases duodenal biopsy was performed. Altogether 85 ultrasonic examinations of the abdomen were performed on 58  children with coeliac disease. Twenty-seven children were in process of the initial diagnosis (IgA EmA positive  and villous atrophy (VA)  III-IV degree). In 20 children a characteristic picture of the abdomen was observed with overflowing liquid contents in the lumen ( pseudoascites)  and meteorismus. ( Suoliäänet vilkaat ja loiskivat).  On gluten challenge ( 7 children), in all cases we noticed changes in the ultrasonographic picture of the intestines.  Conclusions: (1) Enlarged intestinal loops with  a considerable amount of liquid contents in the lumen and live peristalsis seen in ulttrasound examination may be a symptom of malabsorption syndromes and should accelerate gastroenterological diagnosis.  (2) Appearance of such a picture of intestines in ultrasonic examination in children being treated by gluten free diet may be  a first sign of not properly observing gluten-free diet. (3)  During gluten provocation the changes in the ultrasonographic picture of intestines may preceed the appearane of antiendomysial antibodies (anti-EmA) .

A41
SOME PARAMETERS OF SERUM LIPID PROFILE IN CHILDREN WITH COELIAC DISEASE
Obuchowicz A, Marek M, Kalita B, Slimok M, Z Szcepanski. Silesian Academy of Medicine,  Bytom,, Poland

 A42
THE USEFULNESS OF BETA-2 MICROGLOBULIN DETERMINATION IN CHILDREN WITH COELIAC DISEASE
Beta-2-m levels were raised , indicating indirectly the state of intestinal mucosa and the changes caused in it by glutren. Beta-2-m determiantion can be useful as a non-invasive means of monitoring treatment with a gluten-free diet. 
 Sikora A, Kalita B, Bukowska C,  Slimok M,  Szczepanski Z. , Sulej J, Chorzelski TP

A43
ABNORMAL RECTOSIGMOID MOTILITY IN UNTREATED CELIAC DISEASE: IMPROVEMENT ON GLUTEN-FREE DIET:
Low levels of TG and VLDL in all children  with CD.
 In children   with CD ,correctly observing a gluten-free diet, both total cholesterol levels and arterosclerosis risk factor are higher as compared to children with CD  and not observing a gluten free diet and children  without CD. 
Iantorno G, Bilder C, Pedreira S, Niveloni S, Smecuol E, Vazquez H, Mazure R, Boerr L, Maurino E, Bai JC.

A44
PERINUCLEAR ANTINEUTROPHIL CYTOPLASMIC ANTIBODIES (p-ANCA) IN CELIAC DISEASE (CS)
Freeman HJ.  University of British Columbia, Vancouver, Canada. 

A45
SCANNING ELECTRON MICROSCOPY IN COELIAC DISEASE
Magliocca FM, Bonamico M, Petrozza V, Mariani P, MontuoAi M, Carpino F. Rome, Italy

A46
HELICOBACTER PYLORI INFECTION IN COELIAC CHILDREN
Slimok M, Sikora A, Kalita B, Zdmudzinzka-Kitczak J, Stoltny L, Szczepanski Z. Bytom,, Poland

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